ASEA REDOX Gene Study Summary
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(pdf - EN) |
White Paper on
In-Vitro Bioactivity of ASEA™
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(pdf - EN) |
Report for ASEA on VO2max Athletic Endurance Enhancement Testing
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(pdf - EN) |
Klinisch getest door Dermatest®, een internationaal gerenommeerd dermatologisch onderzoeksinstituut,
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Het felbegeerde 5-sterren zegel |
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Am J Physiol v275 pC1-C24 (1998), “Interaction of reactive oxygen species with ion transport mechanisms (an invited review)”, Joseph I. Kourie (http://www.ncbi.nlm.nih.gov/pubmed/9688830).
Arteriosclr Throm. Vasc. Biol., v20, p2175-2183 (2000), “Modulation of Protein Kinase Activity and gene expression by reactive oxygen species and their role in vascular physiology and pathophysiology”, Kathy K. Griendling, et al. (http://www.ncbi.nlm.nih.gov/pubmed/11031201).
J of Medical Microbio, v52, p643-651 (2003), “Reassessment of the microbicidal activity of reactive oxygen species and hypochlorous acid with reference to the phagocytic vacuole of the neutrophil granulocyte”, Emer P. Reeves (http://www.ncbi.nlm.nih.gov/pubmed/12867557).
The J. of Cell Biology (JCB), v155(6), p1003-15 (Dec 2001), “VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release”, M. Madesh, G. Hajonczky (http://www.ncbi.nlm.nih.gov/pubmed/11739410).
Genes and Development, v15, p2922-2933 (2001), “The expanding role of mitochondria in apoptosis”, X. Wang (http://www.ncbi.nlm.nih.gov/pubmed/11711427).
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